Conductor losses calculation in two-dimensional simulations of H-plane rectangular waveguides

This paper presents a novel numerical approach to simulate H-plane rectangular-waveguide microwave circuits considering a reduced quasi-2D simulation domain with benefits for computational cost and time. With the aim to evaluate the attenuation of the full height 3D component, we propose a modified expression for the waveguide top/bottom wall conductivity. Numerical 2D simulations are validated against results from full wave 3-D commercial electromagnetic simulator. After a benchmark on a simple straight waveguide model, the method has been successfully applied to an asymmetric un-balanced power splitter, where an accurate power loss prediction is mandatory. Simulation time and memory consumption can be reduced by a factor ten and seven respectively, in comparison with complete 3D geometries. Finally, we show that, also for quasi-2D E-bend waveguide, a case where the translational H-plane symmetry is broken, the error on conductor losses computation is mitigated by our approach since the method remains still valid in a first approximation

STOMATAL CARPENTER 1 CONTROLS STOMATA DEVELOPMENT BY AFFECTING SPEECHLES ACTIVITY

Stomata consist of two specialised epidermal cells termed guard cells (GCs) surrounding a pore, through which gas exchange can occur. In Arabidopsis thaliana, a dedicated cell lineage is initiated and undergoes a series of cell divisions to produce a stoma. A set of basic helix-loop-helix (bHLH) transcription factors regulates the differentiation events through the lineage. The initiation and proliferation of stomatal lineage cells is controlled by the transcription factor SPEECHLESS (SPCH), which drive the asymmetric division of meristemoid mother cell to originate a small triangular cell termed meristemoid. Which transcriptional events precede stomata lineage specification and are required for correct GC patterning, however, remain unclear. The DOF transcription factor STOMATAL CARPENTER 1 (SCAP1) has previously shown to be expressed in GC and involved in stomata function, by activating a set of GC\u2013specific genes required for GC maturation and activity. We show that SCAP1 expression can also be observed in young leaf primordia, before any GC differentiation occurs. The study of transgenic plants carrying a proSCAP1:GUS-GFP transcriptional fusion coupled with quantitative PCR analyses indicate that SCAP1 expression is maximal in a stomatal lineage competence domain, coincident with SPCH expression. We found that SCAP1 modulates stomata development; independent scap1 loss-of- function mutants show a reduced number of GCs whilst SCAP1 over expression lines have an increased number of GCs in addition to altered GC distribution and spacing patterns. Confocal imaging of SPCH-GFP protein in a background carrying inducible SCAP1 shows that SCAP1 activation results in an increased number of nuclei expressing SPCH-GFP. Our results suggest an early role for SCAP1 in GC differentiation through SPCH protein stabilization. SCAP1 may thus link different aspects of GC biology including specification, maturation and function

The role of brassinosteroids in controlling plant height in poaceae : a genetic perspective

The most consistent phenotype of the brassinosteroid (BR)-related mutants is the dwarf habit. This observation has been reported in every species in which BR action has been studied through a mutational approach. On this basis, a significant role has been attributed to BRs in promoting plant growth. In this review, we summarize the work conducted in rice, maize, and barley for the genetic dissection of the pathway and the functional analysis of the genes involved. Similarities and differences detected in these species for the BR role in plant development are presented. BR promotes plant cell elongation through a complex signalling cascade that modulates the activities of growth-related genes and through the interaction with gibberellins (GAs), another class of important growth-promoting hormones. Evidence of BR\u2013GA cross-talk in controlling plant height has been collected, and mechanisms of interaction have been studied in detail in Arabidopsis thaliana and in rice (Oryza sativa). The complex picture emerging from the studies has highlighted points of interaction involving both metabolic and signalling pathways. Variations in plant stature influence plant performance in terms of stability and yield. The comprehension of BR\u2019s functional mechanisms will therefore be fundamental for future applications in plant-breeding programs

Hybrid neutron stars within the Nambu-Jona-Lasinio model and confinement

Recently, it has been shown that the standard Nambu-Jona-Lasinio (NJL) model is not able to reproduce the correct QCD behavior of the gap equation at large density, and therefore a different cutoff procedure at large momenta has ben proposed. We found that, even with this density dependent cutoff procedure, the pure quark phase in neutron stars (NS) interiors is unstable, and we argue that this could be related to the lack of confinement in the original NJL model.Comment: 2 pages, 1 figure, to be published in the proceedings of the conference EXOCT07, Catania, 11-15 June, 200

Noncommutativity and Lorentz Violation in Relativistic Heavy Ion Collisions

The experimental detection of the effects of noncommuting coordinates in electrodynamic phenomena depends on the magnitude of |\theta B|, where \theta is the noncommutativity parameter and B a background magnetic field. With the present upper bound on \theta, given by \theta_{\rm bound} \simeq 1/(10 {\rm TeV})^2, there was no large enough magnetic field in nature, including those observed in magnetars, that could give visible effects or, conversely, that could be used to further improve \theta_{\rm bound}. On the other hand, recently it has been proposed that intense enough magnetic fields should be produced at the beginning of relativistic heavy ion collisions. We discuss here lepton pair production by free photons as one kind of signature of noncommutativity and Lorentz violation that could occur at RHIC or LHC. This allows us to obtain a more stringent bound on \theta, given by 10^{-3} \theta_{\rm bound}, if such "exotic" events do not occur.Comment: Five pages, no figures

PACAP and VIP Mitigate Rotenone-Induced Inflammation in BV-2 Microglial Cells.

Rotenone is a commercial pesticide commonly used to model Parkinson's disease (PD) due to its ability to induce dopaminergic degeneration. Studies have confirmed that rotenone causes microglial activation, which seems to contribute to the toxic effects seen in rodent models. Pituitary adenylate cyclase-activating polypeptide (PACAP) and vasoactive intestinal peptide (VIP) are two structurally related neuropeptides that have robust neuroprotective and anti-inflammatory properties. However, their ability to regulate microglial activity in response to rotenone is not fully understood. Using rotenone as an inflammatory stimulus, we tested whether PACAP or VIP could mitigate microglial activation in BV2 microglial cells. Rotenone dose-dependently reduced cell viability and the percentage of apoptotic cells. It also increased the release of nitric oxide (NO) in culture media and the expression of microglial activation markers and pro-inflammatory markers, including CD11b, MMP-9 and IL-6, and heightened the endogenous levels of PACAP and its preferring receptor PAC1. Co-treatment with PACAP or VIP prevented rotenone-induced increase of NO, CD11b, MMP-9 and IL-6. These results indicate that both PACAP and VIP are able to prevent the pro-inflammatory effects of rotenone in BV2 cells, supporting the idea that these molecules can have therapeutic value in slowing down PD progression